Graves’ disease is an autoimmune thyroid disease in which stimulating antibodies against the thyroid stimulating hormone receptor cause hyperthyroidism. With appropriate treatment, we can establish a normal serum concentration of thyroid hormones and thereby control the disease. Thyroid hormones have both genomic and non-genomic effects on cardiomyocytes and affect the contractile function of the heart. Hyperthyroidism can increase the risk of sinus tachycardia and atrial fibrillation (AF), and, through direct or indirect mechanisms, it can also cause or worsen heart failure. A severe, potentially fatal complication of hyperthyroidism is tachycardic dilated cardiomyopathy, which affects approximately 1% of patients. With timely treatment, when the euthyroid state is achieved, cardiac function can improve or even normalize. The presented clinical case shows a female patient with newly diagnosed Graves’ disease, AF with a tachycardic ventricular response, and consequent tachycardic dilated cardiomyopathy
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