This thesis consists of presenting the results of certain studies of yeast aging as a model organism. Due to the increasing share of the old population, aging is becoming a topic, which is being increasingly explored for the purpose of a better and longer life. Aging can be influenced by genetic modifications, such as shutting off genes or gene deletion, treatment with chemical compounds and with a change in diet, where all modes affect pathways, that sense nutrient availability. The first part of the thesis presents why yeast can be a suitable model for the study of aging processes and then replicative (RLS) and chronological (CLS) aging are explained. This is followed by an explanation of the processes that occur through RLS and CLS, depending on the activaton/deactivation of pathways that detect nutrient availability.
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